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Abstract
Persistent SARS-CoV-2 antigen is proposed as a key factor in Long COVID.
- Post-acute sequelae of SARS-CoV-2 infection may be linked to impaired antibody function.
- Inefficient clearance of infected cells and viral antigen could be due to altered Fc-mediated antiviral activity.
- Differences in IgG1 Fc fucosylation are examined as a potential regulator of antibody function.
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