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Abstract
Aging is associated with compromised mitochondrial function, which can lead to excessive production of reactive oxygen species and increased inflammation.
- Impaired mitochondrial function in aging organs may contribute to cellular senescence and chronic diseases.
- Regular physical exercise is associated with the preservation of mitochondrial integrity.
- Exercise may enhance mitochondrial regulation across various organs.
- Understanding the molecular mechanisms of exercise can elucidate its potential anti-aging effects.
- Targeted lifestyle modifications could help restore mitochondrial homeostasis and mitigate degenerative functional decline.
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