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Abstract
The prevalence of atrial fibrillation (AF) is increasing rapidly with global aging.
- Cellular senescence is a key factor contributing to the development of atrial fibrillation.
- The mechanisms driving atrial cardiomyocyte senescence are not fully understood.
- MAPK15 has been linked to regulating cell senescence in epithelial cells, but its role in heart cells is unclear.
- This investigation focuses on the relationship between MAPK15, atrial cardiomyocyte senescence, and atrial fibrillation.
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