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Abstract
Sepsis-induced acute kidney injury (S-AKI) is characterized by tubular damage, inflammation, and dysregulated autophagy.
- N6-methyladenosine (m6A) RNA modification plays a role in regulating mRNA stability and responses to cellular stress.
- The specific mechanisms of m6A modification in the context of S-AKI are not fully understood.
- Further investigation is needed to clarify how m6A might influence the pathology of S-AKI.
Simplified