Molecular biology reports

Nicotinamide mononucleotide may reduce testicular cell death and improve mitochondrial function in rats treated with doxorubicin

Updated

Abstract

Doxorubicin treatment is associated with testicular toxicity in male rats.

  • Chemotherapy with doxorubicin may lead to increased testicular apoptosis.
  • Inflammatory changes are observed in the testes of rats treated with doxorubicin.
  • Oxidative stress is a potential consequence of doxorubicin exposure in testicular tissue.
  • Nicotinamide mononucleotide (NMN) is being investigated for its ability to protect against these effects.
  • Improved mitochondrial function is a proposed mechanism for NMN's protective role.

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Funding

Competing interests

0 of 5
authors report competing interests
5 report none
PubMed

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