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Abstract
Ferroptosis and cuproptosis are two types of cell death that significantly influence gastric cancer progression.
- Ferroptosis is marked by iron-induced damage to lipids, while cuproptosis is linked to copper-related mitochondrial dysfunction.
- The interaction between ferroptosis and cuproptosis in gastric cancer involves oxidative stress and disruptions in cellular energy production.
- This interplay affects processes such as autophagy and the balance of glutathione, a critical antioxidant.
- Identifying the mechanisms of this crosstalk may reveal new therapeutic targets for gastric cancer treatment.
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