International review of neurobiology

Gut bacteria imbalance linked to lower brain chemical levels in Parkinson's disease

Updated

Abstract

Intestinal dysbiosis is suggested to play a critical role in Parkinson's Disease progression.

  • Parkinson's Disease is associated with motor and non-motor symptoms.
  • Pathophysiological mechanisms of PD include alpha-synuclein aggregation, mitochondrial dysfunction, neuroinflammation, and oxidative stress.
  • Dysbiosis may impact intestinal barrier function and contribute to neurotransmitter deficiencies, including dopamine, serotonin, and GABA.
  • Current treatments involve levodopa and dopamine agonists, alongside gut health therapies like probiotics and Fecal Microbiota Transplantation.
  • Future therapeutic directions may include synbiotics, engineered microbes, phage therapy, and the use of machine learning and artificial intelligence.
  • Preventive strategies may involve lifestyle adjustments and early monitoring of gut health.

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